ARTICLE INFO

Article Type

Original Research

Authors

Saremi   A.T. (*)
Khamesian   M. (1)
Marmazi‎   F. (1)






(*) ‎“Sarem Fertility & Infertility Research Center (SAFIR)” and “Sarem Cell Research Center (SCRC)”, Sarem Women’s Hospital, Tehran, Iran
(1) Sarem Women’s Hospital, Tehran, Iran

Correspondence


Article History

Received:   January  15, 2016
Accepted:   May 14, 2016
ePublished:   June 15, 2017

ABSTRACT

Aims Unexplained asthenospermia is one of the most important causes of male ‎infertility. Biochemical parameters of seminal plasma are useful in diagnosis of ‎this cause of infertility. Three parameters including carnitine, fructose and zinc ‎can indicate the function of epididymis, seminal vesicle and prostate, ‎respectively. The objective of this study was to evaluate the carnitine, fructose ‎and zinc levels in seminal plasma of unexplained asthenospermic patients ‎compared with normal individuals.‎
Materials & Methods In this case-control study, we measured the level of carnitine, fructose and zinc in ‎seminal plasma of 43 unexplained asthenospermic patients (case group) and 37 ‎normal men (control group). Data and demographic characteristics of the patients ‎were collected through a questionnaire. In addition to sperm counts, sperm ‎motility was also evaluated in semen samples. The levels of Zinc, fructose and ‎carnitine were measured by atomic absorption spectrophotometry.‎
Findings There were no significant differences in mean fructose and zinc concentrations in ‎seminal plasma between patient and control groups (p>0.05). Mean concentration ‎of carnitine in the patient and control groups were 181.78±70.14 nmol/ml and ‎‎218.22±58.51 nmol/ml, respectively. This difference was statistically significant ‎‎(p=0.015). Carnitine concentration was positively related to total motility ‎‎(p=0.001) and progressive sperm motility (p=0.001), while there were no ‎significant relationships between zinc and fructose concentrations and sperm ‎motility (p>0.05).‎
Conclusion Reduction of carnitine in unexplained asthenospermic patients due to epididymal ‎disfunction may be the cause of poor sperm motility‎


CITATION LINKS

[1]Freeman EW, Boxer AS, Rickels K, Tureck R, Mastroianni L. Psychological evaluation and support in a ‎program of in vitro fertilization and embryo transfer. Fertil Steril. 1985;43(1):48-53.‎
[2]Zavos PM, Abou-Abdallah M, Aslanis P, Correa JR, Zarmakoupis-Zavos PN. Use of the Multi-ZSC one-step ‎standardized swim-up method: Recovery of high-quality spermatozoa for intrauterine insemination or other ‎forms of assisted reproductive technologies. Fertil Steril. 2000;74(4):834-5.‎
[3]Chen ZJ. Clinical progress in gynecologic reproductive endocrinology. Zhonghua fu chan ke za zhi. ‎‎2009;44(9):655-7. [Chinese]‎
[4]Hanash KA. Book review: Campbell's urology, 7th ed. Ann Saudi med. 1998;18(6):570-1. ‎
[5]Carpino A, Sisci D, Aquila S, Salerno M, Siciliano L, Sessa M, et al. Adnexal gland secretion markers in ‎unexplained asthenozoospermia. Arch Androl. 1994;32(1):37-43. ‎
[6]Check DJ, Check JH, Bollendorf A. Fresh versus frozen seminal plasma for enhancing sperm motility in ‎asthenozoospermic males. Arch Androl. 1991;26(2):79-81.‎
[7]Karacagil M, Sade M, TÜRkyilmaz RK. The effect of zinc, arginine, fructose and seminal supernatant of normal ‎semen on the triple adenosine triphosphatase activities of the spermatozoa from males with ‎oligoasthenozoospermia. Andrologia. 1985;17(4):383-8.‎
[8]Pruneda A, Yeung CH, Bonet S, Pinart E, Cooper TG. Concentrations of carnitine, glutamate and myo-inositol ‎in epididymal fluid and spermatozoa from boars. Anim Reprod Sci. 2007;97(3-4):344-55.‎
[9]Jeulin C, Lewin LM. Role of free L-carnitine and acetyl-L-carnitine in post-gonadal maturation of mammalian ‎spermatozoa. Hum Reprod Update. 1996;2(2):87-102.‎
[10]Kobayashi D, Tamai I, Sai Y, Yoshida K, Wakayama T, Kido Y, et al. Transport of carnitine and acetylcarnitine ‎by carnitine/organic cation transporter (OCTN) 2 and OCTN3 into epididymal spermatozoa. Reproduction. ‎‎2007;134(5):651-8.‎
[11]Andrade-Rocha FT. Seminal fructose levels in male infertility: Relationship with sperm characteristics. Int ‎Urol Nephrol. 1999;31(1):107-11.‎
[12]Henkel R, Bittner J, Weber R, Huther F, Miska W. Relevance of zinc in human sperm flagella and its relation to ‎motility. Fertil Steril. 1999;71(6):1138-43.‎
[13]Andrews JC, Nolan JP, Hammerstedt RH, Bavister BD. Role of zinc during hamster sperm capacitation. Biol ‎Reprod. 1994;51(6):1238-47.‎
[14]Colagar AH, Marzony ET, Chaichi MJ. Zinc levels in seminal plasma are associated with sperm quality in fertile ‎and infertile men. Nutr Res. 2009;29(2):82-8. ‎
[15]Bjorndahl L, Kjellberg S, Kvist U. Ejaculatory sequence in men with low sperm chromatin-zinc. Int J Androl. ‎‎1991;14(3):174-8.‎
[16]Srivastava A, Chopra SK, Dasgupta PR. Biochemical analysis of human seminal plasma. I. Fructose, ascorbate, ‎cholesterol, adenosine triphosphatase and lactic dehydrogenase. Andrologia. 1983;15(5):431-5. ‎
[17]Matalliotakis I, Koumantaki Y, Evageliou A, Matalliotakis G, Goumenou A, Koumantakis E. L-carnitine levels in ‎the seminal plasma of fertile and infertile men: Correlation with sperm quality. Int J Fertil Womens Med. ‎‎2000;45(3):236-40.‎
[18]Burtis CA, Ashwood ER, Bruns DE. Tietz Fundamentals of Clinical Chemistry. 6th ed. Amsterdam: Saunders ‎Elsevier. 2008. P.976.‎
[19]Li K, Li W, Huang YF, Shang XJ. Level of free L-carnitine in human seminal plasma and its correlation with ‎semen quality. Zhonghua Nan Ke Xue. 2007;13(2):143-6.‎